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GG-13 Pathogenesis of lupus proliferative glomerulonephritis (LPGN): podocytes as targets and responders provide evidence for the importance of local factors controlling end organ damage
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Authors

  • Shu Man Fu 1Division of Rheumatology and Immunology, the Centre of Inflammation, Immunity and Regenerative Medicine and Division of Nephrology, Department of Medicine, University of Virginia, Charlottesville, VA, USA PubMed articlesGoogle scholar articles
  • Hongyang Wang 1Division of Rheumatology and Immunology, the Centre of Inflammation, Immunity and Regenerative Medicine and Division of Nephrology, Department of Medicine, University of Virginia, Charlottesville, VA, USA PubMed articlesGoogle scholar articles
  • Chao Dai 1Division of Rheumatology and Immunology, the Centre of Inflammation, Immunity and Regenerative Medicine and Division of Nephrology, Department of Medicine, University of Virginia, Charlottesville, VA, USA PubMed articlesGoogle scholar articles
  • Sun-sang J Sung 1Division of Rheumatology and Immunology, the Centre of Inflammation, Immunity and Regenerative Medicine and Division of Nephrology, Department of Medicine, University of Virginia, Charlottesville, VA, USA PubMed articlesGoogle scholar articles
  • Felicia Gaskin 2Department of Psychiatry and Neurobehavioral Sciences University of Virginia, Charlottesville, VA, USA PubMed articlesGoogle scholar articles

Citation

Fu SM, Wang H, Dai C, et al
GG-13 Pathogenesis of lupus proliferative glomerulonephritis (LPGN): podocytes as targets and responders provide evidence for the importance of local factors controlling end organ damage

Publication history

  • First published August 31, 2016.
Online issue publication 
August 31, 2016

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